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Bibliographical entry (without author) : | Neonatal Morbidity associated with uterine rupture: what are the risk factors?. AJOG, 186, p.311 |
Author(s) : | Bujold |
Year of publication : | 2002 |
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Résumé (français) : |
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Abstract (English) : | Retro., 2233 TOL had 23 cases of uterine rupture after a previous LTCS. Nine infants (39.1%) had severe acidosis (pH <7.0), among these, 3 neonates had severe hypoxic-ischemic encephalopathy and another neonate died. Placental or fetal extrusion or both were associated with severe20metabolic acidosis but not with other factors (birth weight, induction of labor, use of oxytocin, epidurals and cervical dilatation). Two newborns with severe acidosis had impaired motor development even with an intervention time less than 18 minutes from the onset of prolonged deceleration to delivery. Conclusion: When uterine rupture occurs, placental or fetal extrusion was the most important factor associated with severe metabolic acidosis; Prompt intervention did not always prevent severe metabolic acidosis and neonatal morbidity. |
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Resumen (español) : |
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Comments : | Fiche importée de http://www.worldserver.com/turk/birthing/rrvbac2000-4.html avec l’aide de Ken Turkowski, septembre 2005 |
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Keywords : | ➡ vaginal birth after caesarean ; c-section/caesarean ; induction of labor ; post-term pregnancy |
Author of this record : | Ken Turkowski — 01 Feb 2006 |
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